30 Bananas a Day!

Hey folks

I got my blood results back a little while ago. I go religiously - every 6 months or so and have been doing so for a decade.

My results go as follows: 16/02/11 triglyceride = 0.6 (they need to be <1.5 apparently). 28/09/11 = 0.7. Then I started HCV (not entirely raw, I have been eating steamed rice and steamed sweet potatoes some days. I drink 3-4 litres of water per day, and get 2700-3000 calories a day). Anyway, on the 09/02/2012 triglycerides came back as 1.2. So, within about three or so months of eating this way, the tri's have spiked 42%.  I'm not freaking out or anything, I just wanted to ask what the reason was behind it.

I read somewhere that there is a direct link between high tri's and low vit d levels. My vit D levels have been in the 80's (85, 83) and this time it came back as 72 (the 'normal' is >75). I see that as a minor drop in vit d for such a great jump in tri's but who am I to say?

Any feedback or info would be greatly appreciated. Thanks for taking the time to read.

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My HbA1c is within normal range.   The lab blood tests show fasting glucose at 85.   My only complaint regarding blood sugar is a high meter reading (200+) after a banana smoothie.  I have no sugar crashes.

Are the bloodtests done after fasting for 8-12 hours? I believe eating before the tests can cause higher trigs

Yes, every test I've done has been overnight fasting, approx 12 hours.  No breakfast till after the test.

Yep, blood tests are done after fasting.

Genetics can play a part in how your body handles nutrients.

I have been wondering about this as well. I have seen several people experiencing high trigs. But I have also seen long term 811 people with really low trigs. 

To be honest, the idea that eating lots of fruit may cause cardiovascular disease seems to be really counterintuitive.  

I agree.

Also, if the fruit was the cause, it would always lead to high trig's. It would stand to reason that there are other factors involved. Whether or not high fruit consumption even interplays with those other factors is a total unknown.

Lastly, if memory serves me, the study upon which Dr. McDougall based his assertion used several sources of fructose, including fruit juices (probably pasteurized to boot) and other fruit drinks.

No, that is not true. Genetics and function of liver have a role. Remember, not all people are born equal anymore. Just look at the genetic problems we have today. The word always does not fit for every one. 

Like I said::

"It would stand to reason that there are other factors involved. Whether or not high fruit consumption even interplays with those other factors is a total unknown."

My trygs increased on an 811vegan diet but

1.  I'm not 100% raw yet though almost there

2. They are still reasonably low, within limits basically

One interesting thing I've read in  Dr Ornish's book on Reversing Heart Disease

was that even vegans with high trygl. still had no coronary problems, at least compared to
non-vegans with high tryg.  

So it's not a big deal apparently.

THen i've read somewhere else (maybe on this forum) that if you don't eat enough calories,

your body uses fat for energy and by breaking it down, somehow trygs end up in the blood

where they are picked up in the test, so it's recommended to eat more.

I wonder if my levels improved now that i eat more

but i'll wait another while before testing again.


Here is a research article of Ornish. I had a very very quick read, but he says this:

"High-density lipoprotein levels de- creased and triglycerides increased in experimental group patients overall, al- though the ratio of LDL to HDL was improved. Recent reports assert that this phenomenon, which is often seen in very low-fat diets, may be harmful.22,23 However, patients in the Lifestyle Heart Trial showed even more regres- sion of coronary atherosclerosis after 5 years than after 1 year as well as significantly decreased cardiac events"

and :

Populations consuming low-fat, plant-based diets have low HDL choles- terol levels and low rates of coronary heart disease. Our data provide evidence using quantitative coronary arteriogra- phy in this population that diet-induced lowering of HDL cholesterol does not confer the same risk of atherosclerosis as do low HDL cholesterol levels in Americans consuming a high-fat diet.25


"Experimental group patients whose tri- glycerides increased during the first year were asked to minimize their in- take of simple carbohydrates, and tri- glyceride levels decreased between year 1 and year 5." 


What is considered a simple carb? 



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